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Since most of these processes damage DNA, there are various DNA repairmechanisms to correct the damage incurred and p53 has been shown to play animportant role in several of these repair mechanisms including nucleotide excisionrepair (bulky DNA adducts) [3, 49], base excision repair (base modications) [ 56,100], mismatch repair (base mismatch due to replication errors) [15, 41], homolo-gous recombination repair and non-homologous end joining (DNA double strandbreaks). This control by Tregs was mentioned in the previous section duringthe discussion of the two main effector mechanisms (perforin/granzyme and FAS/FASL) usedby T cells, NK cells, and Tregs

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